Many cases of severe combined immunodeficiency result from mutations in adenosine deaminase (an enzyme involved in nucleic acid turnover). Deficiency of this enzyme's activity leads to a 100-fold increase in dATP, yet also causes a deficiency in dNTPs required by lymphocytes for replication and proper immune function. Explain.

The deficiency in dNTPs is typically caused as a result of mutations such as changes in the adenosine deaminase gene . The adenosine deaminase gene is vital for the production of enzymes commonly found in lymphocytes. The lymphocytes are vital components of the body immune system that fight infections and keep the body healthy.

Explanation:

The deficiency in dNTPs is typically caused as a result of mutations such as changes in the adenosine deaminase gene . The adenosine deaminase gene is vital for the production of enzymes commonly found in lymphocytes. The lymphocytes are vital components of the body immune system that fight infections and keep the body healthy.

his research revealed two genetically different groups of prokaryotes, which justified splitting kingdom monera into two kingdoms. the genetic and cellular differences were so great, that these two kingdoms were eventually placed into different domains in the current three-domain system.

white blood cells or wbc s are mainly 2 types-

1.agranulocytes

2.granulocytes

the monocytes or macrophages of agranulocytes , dendritic cells which are accessory leucocytes, and b cells which is one type of lymphocytes   identify the antigens present on the pathogen's surface. these three cells are called antigen presenting cells(apc) which are phagocytes and destroy the antigens by secreting antitoxins. besides this leukocyte is always plays vital role in the immunity of the body.